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Background
Low‐normal thyroid function within the euthyroid range has been suggested to enhance atherosclerosis susceptibility. Paraoxonase‐1 (PON‐1) may protect against atherosclerotic cardiovascular disease development by attenuating oxidative stress. We evaluated relationships of PON‐1 with thyroid stimulating hormone (TSH), free T4, free T3, lipids and apolipoprotein (apo)A‐I in euthyroid subjects,...
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