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The voltage-gated cardiac sodium channel, SCN5A, is responsible for the initial upstroke of the action potential. Mutations in the humanSCN5Agene cause susceptibility to cardiac arrhythmias and sudden death in the long QT syndrome (LQT). In this report we characterize the genomic structure ofSCN5A. SCN5Aconsists of 28 exons spanning approximately 80 kb on chromosome 3p21. We describe the sequences...
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