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Aims
It has been demonstrated that neuroinflammation is associated with cardiovascular dysfunction. The phosphoinositide‐3 kinase (PI3K) signaling in the rostral ventrolateral medulla (RVLM), a key region for sympathetic outflow, is upregulated and contributes to increased blood pressure (BP) and sympathetic outflow in hypertension. This study was designed to determine the role of the PI3K signaling...
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