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Skeletal muscle contraction is basically controlled by Ca2+ release and its reuptake into the sarcoplasmic reticulum. However, the long-term maintenance of muscle function requires an additional Ca2+ influx from extracellular. Several mechanisms seem to contribute to the latter process, such as store-operated Ca2+ entry, stretch-activated Ca2+ influx and resting Ca2+ influx. Candidate channels that...
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