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In the present study, we propose a mathematical model to describe the key role of postsynaptic calcium concentration in Late Long-Term Potentiation (L-LTP) induction. This model is developed based on physiological facts of a typical CA3-CA1 tripartite synapse. In the proposed model, each astrocyte has a bidirectional signaling with pre synaptic neuron and based on its intracellular calcium oscillations...
In this work we propose a model to describe impairment of Long-Term Potentiation (LTP) due to Amyloid Beta (Aβ) accumulation during Alzheimer's Disease (AD) in hippocampus. The model embeds astrocyte-neuron interactions and Spike Timing-Dependent Plasticity (STDP) in CA3-CA1 synapses. In our proposed model, considering physiological facts, each astrocyte has a bidirectional signaling with four neurons,...
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