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Although imatinib (IM) has been demonstrated to be an efficient treatment in chronic myeloid leukemia (CML), some patients still experience IM resistance and disease relapse. Through in vitro studies, we observed that HDAC3 levels were elevated in BCR‐ABL1 kinase−independent, IM‐resistant primary cells from CML patients and in IM‐resistant K562 (K562R) cells and that downregulation of HDAC3 could...
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