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Complement components and their receptors are found within and around amyloid β (Aβ) cerebral plaques in Alzheimer's disease (AD). Microglia defend against pathogens through phagocytosis via complement component C3 and/or engagement of C3 cleavage product iC3b with complement receptor type 3 (CR3, Mac‐1). Here, we provide direct evidence that C3 and Mac‐1 mediate, in part, phagocytosis and clearance...
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