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Pemphigus vulgaris (PV) is a potentially life‐threatening autoimmune disease of the skin and mucous membranes. Its pathogenesis is based on IgG autoantibodies that target the desmosomal cadherins, desmoglein 3 (Dsg3) and desmoglein 1 (Dsg1) and induce intra‐epidermal loss of adhesion. Although the PV pathogenesis is well‐understood, therapeutic options are still limited to immunosuppressive drugs,...
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