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Dissemination of cancer cells from primary to distant sites is a complex process; little is known about the genesis of metastatic changes during disease development. Here we show that the metastatic potential of E2F1‐dependent circulating tumour cells (CTCs) relies on a novel function of the hyaluronan‐mediated motility receptor RHAMM. E2F1 directly up‐regulates RHAMM, which in turn acts as a co‐activator...
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