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Slow delayed-rectifier potassium current (IKs) channels, made of the pore-forming KCNQ1 and auxiliary KCNE1 subunits, play a key role in determining action potential duration (APD) in cardiac myocytes. The consequences of drug-induced KCNQ1 splice alteration remain unknown.To study the modulation of KCNQ1 alternative splicing by amiloride and the consequent changes in IKs and action potentials (APs)...
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