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Antibodies raised against human α2-6 and β2-4 nicotinic receptor subunits were utilized to fractionate 3 H-epibatidine binding in human temporal cortex and striatum. The predominant receptor subtypes in both regions contained α4 and β2 subunits. In normal cortex, 10% of binding was also associated with α2 subunits, whereas in the striatum, contributions by α6 (17%) and β3 (23%) were observed...
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