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The neuropathological hallmarks of Alzheimer’s disease (AD) and other taupathies include neurofibrillary tangles and plaques. Despite the fact that only 2–10% of AD cases are associated with genetic mutations, no nontrasgenic or metabolic models have been generated to date. The findings of tryptophanyl-tRNA synthetase (TrpRS) in plaques of the AD brain were reported recently by the authors. Here it...
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