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We performed genotyping and exon‐level expression profiling on 21 glioblastomas (GBMs) and 19 oligodendrogliomas (ODs) to identify genes involved in glioma initiation and/or progression. Low‐copy number amplifications (2.5 < n < 7) and high‐copy number amplifications (n > 7) were more frequently observed in GBMs; ODs generally have more heterozygous deletions per tumor. Four high‐copy amplicons...
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