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AbstractDeficiency of the G protein subunit Gi2 that is known to mediate the inhibitory control of adenylylcyclase impairs insulin action [11]. Using the promoter for the phosphoenolpyruvate carboxykinase gene, conditional, tissue-specific expression of the constitutively active mutant form (Q205L) of Gi2 was achieved in mice harboring the transgene. Expression of Q205L Gi2 was detected in liver and...
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