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BackgroundHelicobacter pylori is the primary cause of gastric cancer, but about 9% of cases harbor Epstein–Barr virus (EBV) in the tumor cells. There is limited evidence on the possible interaction or antagonism between these infectious agents in gastric carcinogenesis.
MethodsWe compared H. pylori serologic profiles of EBV‐positive (n = 58) and EBV‐negative (n = 111) noncardia gastric cancer patients...
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