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The purpose of this study was to determine whether activation of ATP-sensitive K + (K ATP ) channels with diazoxide (DIZ) is able to prevent the cleavage of cytosolic μ-calpain and abrogate the elevation of nuclear c-Fos and c-Jun protein (c-Fos, c-Jun) expressions after hypoxic–ischemia (HI) in brain. The model of hypoxic–ischemic brain injury (HIBI) was made in the 7-day-old Sprague–Dawley...
This study aimed to clarify the neuroprotective mechanism of electro-acupuncture (EA) preconditioning on hypoxic-ischemic brain injury (HIBI). Using Western blot, the expression of c-fos protein (c-Fos) and c-jun protein (c-Jun) induced by glibenclamide, an ATP-sensitive potassium (K ATP ) channel blocker was examined from cerebral cortical and hippocampal samples in neonatal hypoxic-ischemic...
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