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Tumor necrosis factor-alpha (TNF-α) is a cytokine that may contribute to the pathogenesis of septic shock, rheumatoid arthritis, cancer, and diabetes. Prostaglandins endogenously produced by macrophages act in an autocrine fashion to limit TNF-α production. We investigated the timing and signaling pathway of prostaglandin-mediated inhibition of TNF-α production in Raw 264.7 and J774 macrophages. TNF-α...
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