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The intracellular TRIM5α protein successfully inhibits HIV-1 infection in rhesus monkeys, but not in humans [1–6]. A few amino acids in the virus-interacting SPRY domain [7] were found to be responsible for most of this anti-viral specificity [8–10], raising the possibility that genetic variation among humans could result in TRIM5α proteins with a spectrum of potencies. We found several nonsynonymous...
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