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Tumor‐draining lymph nodes (TDLNs) are the first sites where tumor components reach and dendritic cells (DCs) present tumor‐associated antigens to T cells. DCs rely on autophagy to process tumor antigens into epitope peptides to form epitope‐MHC complexes. Selective delivery of autophagy‐stimulating drugs to TDLNs may be a precise strategy to boost chemotherapy‐induced antitumor immunity. Here, a...
Chemoimmunotherapy‐induced antitumor immune response is highly dependent on tumor autophagy. When tumor cells are treated with chemoimmunotherapy, timely overactivated autophagy can not only lead more tumor cells to death, but also participate in the endogenous antigen presentation and immune stimulators secretion of dying cells, thus plays a vital role. However, timely and accurately overactivated...
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