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Inflammation, an important mechanism in the pathogenesis of atrial fibrillation (AF), can be regulated by CD36 in monocytes.The purpose of this study was to test the hypothesis that CD36 in monocytes contributes to the pathogenesis of AF.A prospective study that enrolled 87 patients with AF and 70 without AF was conducted.Compared to patients without AF, patients with AF had monocytes with a lower...
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