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Immune activation plays a signicant role in the development and progression of chronic heart failure (CHF). Indeed, pro-inammatory cytokines, especially tumour necrosis factor- (TNF) are activated in this condition and exert direct detrimental actions on the myocardium. Physiological dampeners of TNF production, such as interleukin-10, catecholamines, cortisol, and others fail in the course of the...
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