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Gene mutations and functional inhibition are the major obstacles for p53‐mediated oncotherapy. For p53‐wild‐type tumors, the underlying mechanisms of functional inhibition of p53 during oncogenesis are unknown. The results reveal that the expression of the MDM2 inhibitor ARF is inhibited in p53‐wild‐type tumors, indicating that the restoration of ARF could be a potential oncotherapy strategy for p53‐wild‐type...
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