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Hypoxic injury poses a severe threat to cell survival. Activation of pro‐survival autophagy via autophagy‐inducing nanomaterials (AINMs) has emerged as one of the most promising therapeutic strategies. However, internalized AINMs easily bind with intracellular lysosomes, resulting in disruptive effects on autophagic flux and limited pro‐survival effects. Herein, inspired by the naturally existing...
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