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In most human malignant tumors, retinoblastoma tumor‐suppressor gene (RB) product is inactivated by phosphorylation. Therefore, cancer preventive agents or molecular‐targeting agents can inhibit the tumor growth at G1 phase through RB reactivation. However, little is known about the effectiveness of RB reactivating agents against malignancies with mutated RB. We report here that chemopreventive agent...
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