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The replication of HBV in hepatocytes can be effectively inhibited by lifelong antiviral therapy. Because of the long‐term presence of HBV reservoirs, the virus rebound frequently occurs once the treatment is stopped, which poses a considerable obstacle to the complete removal of the virus. In terms of gene composition, regulation of B cell action and function, CXCR5+CD8+ T cells are similar to CXCR5...