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Aim
LDL receptor‐related protein type 2 (LRP2) is highly expressed on both yolk sac and placenta. Mutations in the corresponding gene are associated with severe birth defects in humans, known as Donnai–Barrow syndrome. We here characterized the contribution of LRP2 and maternal plasma cholesterol availability to maternal–fetal cholesterol transport and fetal cholesterol levels in utero in mice.
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