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Cardiac fibroblasts (CF) have direct and potent effects on myocardial remodelling by proliferating, differentiating and secreting extracellular matrix proteins. Prolonged activation of CF leads to cardiac fibrosis and reduces myocardial contractile function. In previous studies we showed that 2,3,4′,5‐tetrahydroxystilbene‐2‐O‐β‐d‐glucoside (TSG) exerts cardiac protection, but the mechanism involved...
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