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We have investigated the neuroprotective effect of sesaminol glucosides (SG) in SK-N-SH cells. SG prevented apoptotic cell death induced by Aβ25–35. In parallel, SK-N-SH cells exposed to Aβ25–35 underwent oxidative stress as shown by the elevated level of intracellular ROS, lipid peroxidation, and 8-hydroxy-2′-deoxyguanosine (8-OHdG) formation, which were effectively suppressed by SG treatment. Furthermore,...
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