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The phenomenon of imatinib resistance in Chronic Myeloid Leukemia (CML) can be mediated by “BCR-ABL fusion transcript dependent” and “BCR-ABL fusion transcript independent” pathways. The main means of “BCR-ABL fusion transcript dependent” pathway, responsible for 40–90% of such cases, is the occurrence of Kinase Domain (KD) mutations. The aim of this study was to evaluate the commonly occurring KD...
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