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Ischemia followed by reperfusion is known to produce gastric lesions due to oxidative stress, but the role of gastric H + secretion in the formation of this mucosal injury remains unknown. We studied alterations in gastric acid secretion and gastric histamine content, as well as the expression of histidine-decarboxylase and interleukin-1β during the mucosal recovery from ischemia–reperfusion...
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