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AimTo test a hypothesis attributing arrhythmia in Brugada Syndrome to right ventricular (RV) outflow tract (RVOT) conduction abnormalities arising from Nav1.5 insufficiency and fibrotic change.
MethodsArrhythmic properties of Langendorff‐perfused Scn5a+/− and wild‐type mouse hearts were correlated with ventricular effective refractory periods (VERPs), multi‐electrode array (MEA) measurements of action...
AimQT interval prolongation reflecting delayed action potential (AP) repolarization is associated with polymorphic ventricular tachycardia and early after depolarizations potentially initiating extrasystolic APs if of sufficient amplitude. The current experiments explored contributions of altered re‐excitation thresholds for, and conduction of, such extrasystolic APs to arrhythmogenesis in Langendorff‐perfused,...
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