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Therapy‐induced senescence in cancer cells is an irreversible antiproliferative state, which inhibits tumor growth and is therefore a potent anti‐neoplastic mechanism. In this study, low doses of Abrus agglutinin (AGG)‐induced senescence through autophagy in prostate carcinoma cells (PC3) and inhibited proliferation. The inhibition of autophagy with 3‐methyl adenine reversed AGG‐induced senescence,...
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