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One of the major neuropathological hallmarks of Alzheimer's disease (AD) is the deposition of amyloid β‐protein (Aβ) in the brain. Aβ accumulation seems to arise from an imbalance between Aβ production and clearance. Neprilysin (NEP) and insulin‐degrading enzyme (IDE) are the important Aβ‐degrading enzymes in the brain, and deficits in their expression may promote Aβ deposition in patients with sporadic...
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