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We previously showed that recessive ataxic tottering-6j mice carried a base substitution (C-to-A) in the consensus splice acceptor sequence linked to exon 5 of the α 1 subunit of the Cav2.1 channel gene (Cacna1a), resulting in the skipping of exon 5 and deletion of part of the S4–S5 linker, S5, and part of the S5–S6 linker in domain I of the α 1 subunit of the Cav2.1 channel. However,...
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