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A constitutive null mutation of the neural-specific isotype of protein kinase C (γ-PKC) in mice produces alterations in behavioral traits and responses to ethanol suggesting that γ-PKC-mediated phosphorylation is essential for regulation of some behaviors. However, it is possible that some of the effects of γ-PKC gene deletion also may be due to altered gene expression. To examine alterations in gene...
To investigate the contribution of the PKCγ isoform of protein kinase C (PKC) in neurochemical pathways regulating anxiety, mice lacking the gene encoding PKCγ were tested with heterozygote and wild-type littermates in three approach-avoidance tests of anxiety. Null mutant mice consistently displayed a decrease in baseline anxiety-related behaviors in the elevated plus-maze, the black/white box, and...
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