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AbstractSKF 96365, a receptor-mediated Ca2+ entry inhibitor, has been reported to inhibit Ca2+ res- ponses to various agonists without affecting internal Ca2+ release and phosphoinositide turnover. Since muscarinic acetylcholine receptor-mediated phospho- inositide turnover shows a marked dependence on factors affecting cytosolic Ca2+ concentration, the effects of SKF 96365 on the coupling of muscarinic...
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