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To extend current knowledge on the underlying mechanisms of air pollution particulate matter (PM2.5)‐induced human lung toxicity, the metabolic activation of polycyclic aromatic hydrocarbons (PAH) within PM2.5 and PAH–DNA bulky stable adduct patterns in human alveolar macrophage (AM) and/or human lung epithelial L132 cells in mono‐ and cocultures were studied. In the coculture system, only human AM...
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