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Normal cardiac excitation involves orderly conduction of electrical activation and recovery dependent upon surface membrane, voltage‐gated, sodium (Na+) channel α‐subunits (Nav1.5). We summarize experimental studies of physiological and clinical consequences of loss‐of‐function Na+ channel mutations. Of these conditions, Brugada syndrome (BrS) and progressive cardiac conduction defect (PCCD) are associated...
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