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Cerebral vasospasm (CVS) is the most treatable component of subarachnoid hemorrhage (SAH), which can be reduced by endothelin receptor antagonists. Endothelin-evoked vasospasm is considered to be mediated by Ca 2+ influx in the smooth muscle through voltage-dependent Ca 2+ channel (VDCC) and nonselective cation channels (NSCC). Because VDCC antagonists such as nimodipine have been...
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