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This article has been retracted at the request of the Editor-in-Chief and Authors. Please see http://www.elsevier.com/locate/withdrawalpolicy.Reason: Questions have arisen concerning the validity of data in this article. Detailed analysis and comparison of the original and published data revealed that there were discrepancies between the original data and the published figures of Figs. 2a, 3a, 3b,...
Hepatitis C virus (HCV) is efficient in the establishment of persistent infection. We have previously shown that HCV core protein inhibits T cell proliferation through its interaction with the complement receptor, gC1qR. Here we show that HCV core-induced inhibition of T cell proliferation involves a G 0 /G 1 cell cycle arrest, which is reversible upon addition of anti-gC1qR antibody...
Hepatitis C virus (HCV) is remarkable for its ability to establish persistent infection. Studies suggest that HCV core protein modulates immune responses to viral infection and can bind Fas receptor in vitro. To further examine the role of HCV core protein in Fas signaling, full-length (aa 1-192) and truncated (aa 1-152) HCV core proteins were expressed in Jurkat lymphocytes and cells were assayed...
Hepatitis C virus (HCV) is a major human pathogen causing mild to severe liver disease worldwide and is remarkably efficient at establishing persistent infections. Previously, we have shown that the core protein has an immunomodulatory function including the suppression of T lymphocyte responses to viral infection. To investigate the underlying mechanism for the role of core protein in immune modulation,...
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