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Several studies suggest mitochondrial dysfunction as a possible mechanism underlying the development of Alzheimer disease (AD). There is data showing that amyloid-β (Aβ) peptide is present in AD brain mitochondria. The human presequence protease (hPreP) was recently shown to be the major mitochondrial Aβ-degrading enzyme. We investigated if there is an increased susceptibility to AD, which can be...
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