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The extension of axons through the major inhibitory component of the glial scar, chondroitin sulfate proteoglycans (CSPGs), remains a key obstacle for regeneration following spinal cord injury (SCI). We have previously shown that transplants composed of neuronal and glial restricted precursors (NRP and GRP respectively) promote regeneration and connectivity in the injured spinal cord (Bonner et al...
Mice overexpressing mutant α-synuclein develop a progressive loss of motor function associated with the accumulation of aggregated α-synuclein in neurons of the brainstem. Recent reports suggest that tau pathology might also be associated with Parkinson disease (PD) and aggregation of α-synuclein. We now report that mice overexpressing A30P α-synuclein develop abnormally phosphorylated tau in parallel...
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