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Ca2+ has been recognized as a key molecule for chondrocytes, however, the role and mechanism of spontaneous [Ca
2+]
i signaling in cartilaginous extracellular matrix (ECM) metabolism regulation are unclear. Here we found that spontaneous Ca
2+ signal of in‐situ porcine chondrocytes was [Ca
2+]
o dependent, and mediated by [Ca
2+]
i store release. T‐type voltage‐dependent calcium channel (T‐VDCC) mediated...
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