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Objectif de l'etude. - L'hypothese est que l'augmentation de l'expression du gene wild-type p53 pourrait sensibiliser des tumeurs a l'irradiation en favorisant la mort cellulaire programmee, meme dans le cas de tumeurs exprimant wt-p53. Nous avons verifie cette hypothese in vitro dans un modele de cancer de la prostate, puis in vivo sur des souris nude xenogreffees.Materiel et methodes. - Nous avons...
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