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Cardiac sarcoplasmic reticulum (SR) sequesters Ca2+and plays a crucial role in the regulation of intracellular Ca2+. Its functional properties are central to the excitation–contraction (E–C) cycle of cardiac muscle. In this study, we examined the hypothesis that alterations in SR function occur during the development of hypertrophy of the left ventricle (LV) induced in rats by pressure overload secondary...
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