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The capacity of nonsteroidal antiinflammatory drugs (NSAIDs) to prevent prostanoids biosynthesis through the inhibition of COX‐2 enzyme is related to their structural backbone, based on the fusion of a cis‐stilbene unit with a variety of heterocyclic and carbocyclic rings. By this route, a series of new selective COX‐2 inhibitors was developed, by maintaining the 4‐methylsulfone or 4‐methylsulfonamide...
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