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The chemokine CXCL13 has a key role in secondary lymphoid tissue orchestration and lymphoid neogenesis. Transgenic mice deficient in CXCL13 or its receptor CXCR5 have severely impaired lymph node development, lack peritoneal B‐lymphocytes and are deficient in circulating antibodies to common bacterial antigens. However, total circulating numbers of B‐lymphocytes are slightly elevated and humoral responses...
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