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Lrp5, the mutated gene in osteoporosis pseudoglioma (OPPG) and the high bone‐mass syndrome (HBM), regulates bone formation, while β‐catenin, the molecular node of Wnt signaling, regulates bone resorption, suggesting that Lrp5 could act in a Wnt‐independent manner. Using microarray and conditional gene deletion in mice, we showed that Lrp5 actually enhances bone formation by inhibiting the expression,...
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