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Atopic eczema (AE) is characterized by skin barrier and immune dysfunction. Null mutations in filaggrin (FLG), a key epidermal barrier protein, strongly predispose to AE; however, the precise role of FLG deficiency in AE pathogenesis remains incompletely understood.We sought to identify global proteomic changes downstream of FLG deficiency in human epidermal living skin–equivalent (LSE) models and...
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