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Toxic epidermal necrolysis (TEN) represents a rare drug‐induced autoimmune reaction with delayed‐type hypersensitivity that initiates the process of developing massive keratinocyte apoptosis, dominantly in the dermoepidermal junction. Although the etiopathophysiology has not yet been fully elucidated, the binding of Fas ligand (FasL, CD95L) to the Fas receptor (CD95) was shown to play a key role in...
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